Sport Reduces The Risk Of Non-alcoholic Steatosis Of The Liver

Maryam Ayres Author: Maryam Ayres Time for reading: ~2 minutes Last Updated: August 08, 2022
Sport Reduces The Risk Of Non-alcoholic Steatosis Of The Liver

In this article, learn more about Sport Reduces The Risk Of Non-alcoholic Steatosis Of The Liver. Immobility and unhealthy eating are a prerequisite for obesity of the liver ....

American researchers have found that regular exercise reduces the risk of non-alcoholic steatosis, while on the contrary - immobilization is a prerequisite for the so-called obesity of the liver.


Hepatic steatosis, also called fatty liver, is an increasingly common health problem due to a metabolic disorder in which fat is deposited in the liver cells. The normal amount of adipose tissue in the liver is 5-6% of its total mass. A fatty liver can be spoken of when the fat amounts to 25-30% of the mass of the gland.


As a result of fat deposition, hepatocytes begin to have difficulty performing their functions. Because the liver is key to all the biochemical processes in the body, when it is sick, all organs and systems suffer.


Fatty lesions in the liver appear gradually. The speed at which the disease progresses, as well as the extent of the damage, depends on the causes. The most common causes of hepatic steatosis are viral, toxic, genetic, etc.


Hepatic steatosis occurs as a result of congenital or acquired damage to hepatocytes, as well as fat metabolism. Some people are born with a defect that worsens with age and provokes the accumulation of fatty deposits in the liver.


According to American researchers, one of the main reasons for the development of non-alcoholic steatosis of the liver is the lack of sufficient physical activity. Immobilization correlates with obesity, decreased immunity, cardiovascular disease, metabolic syndrome and others.


The findings of researchers at the University of Missouri are based on an experimental study done with laboratory animals.


The experimental mice were divided into several groups, depending on the diet and the ability to move freely and actively expend energy.


In the group in which the mice were not physically active but had free access to food, fatty infiltration of the liver was observed as early as 25 weeks of life. The animals in this group were again divided into two other groups - one continued to live in small cages in which they did not have enough opportunity to move, and the others were subjected to active loads.


A large percentage of laboratory animals with low motor activity developed liver cirrhosis. In others, who were subjected to physical activity and reduced food intake, there was an improvement in the condition.


The study's authors are adamant that exercise and regular physical activity, combined with a healthy, low-fat diet, significantly reduce the risk of nonalcoholic steatosis of the liver.
 

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